1015 Walnut Street
Curtis Building, Suite 611A
Philadelphia, PA 19107
(215) 955-2693
Curtis Building, Suite 611A
Philadelphia, PA 19107
(215) 955-2693
Most Recent Peer-reviewed Publications
- Diagnostic, prognostic, and predictive biomarkers in pancreatic cancer
- Molecular-based and alternative therapies for pancreatic cancer: Looking "out of the box"
- HuR's post-transcriptional regulation of death receptor 5 in pancreatic cancer cells
- Epidermal growth factor receptor and insulinlike growth factor 1 receptor expression predict poor survival in pancreatic ductal adenocarcinoma
- A novel survival-based tissue microarray of pancreatic cancer validates muc1 and mesothelin as biomarkers
- Molecular profiling of synchronous and metachronous cancers of the pancreas reveal molecular mimicry between samples from the same patient
- Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) induces death receptor 5 networks that are highly organized
- Is there a role for the quantification of RRM1 and ERCC1 expression in pancreatic ductal adenocarcinoma?
- Loss of stromal Caveolin-1 expression in malignant melanoma metastases predicts poor surv 1298 ival
- Subject Review: Pancreatic Ductal Adenocarcinoma in the Setting of Mutations in the Cystic Fibrosis Transmembrane Conductance Regulator Gene: Case Report and Review of the Literature
- The Past, Present, and Future of Biomarkers: A Need for Molecular Beacons for the Clinical Management of Pancreatic Cancer
- Molecular profiling of a lethal tumor microenvironment, as defined by stromal caveolin-1 status in breast cancers
- HuR's role in gemcitabine efficacy: An exception or opportunity?
- pp32 (ANP32A) expression inhibits pancreatic cancer cell growth and induces gemcitabine resistance by disrupting HuR binding to mRNAs
- CanScript, an 18-Base pair DNA sequence, boosts tumor cell-specific promoter activity: Implications for targeted gene therapy
- CXCR4 signaling identifies a role for IFT2 in ER-negative breast cancers
- HuR status is a powerful marker for prognosis and response to gemcitabine-based chemotherapy for resected pancreatic ductal adenocarcinoma patients
- Implications of enhancer of zeste homologue 2 expression in pancreatic ductal adenocarcinoma
- Stromal CD10 and SPARC expression in ductal carcinoma in situ (DCIS) patients predicts disease recurrence
- Loss of stromal caveolin-1 expression predicts poor clinical outcome in triple negative and basal-like breast cancers
