1020 Locust Street
Jefferson Alumni Hall, Suite 265
Philadelphia, PA 19107
(215) 503-9983
Jefferson Alumni Hall, Suite 265
Philadelphia, PA 19107
(215) 503-9983
Most Recent Peer-reviewed Publications
- Differential expression of sodium channel β subunits in dorsal root ganglion sensory neurons
- Modulation of Kv3.4 channel N-type inactivation by protein kinase C shapes the action potential in dorsal root ganglion neurons
- Regulatory role of voltage-gated Na + channel β subunits in sensory neurons
- Regulation of Na v1.6 and Na v1.8 peripheral nerve Na + channels by auxiliary β-subunits
- Single-cell analysis of sodium channel expression in dorsal root ganglion neurons
- Y1767C, a novel SCN5A mutation, induces a persistent Na + current and potentiates ranolazine inhibition of Na v1.5 channels
- Role of voltage-gated sodium, potassium and calcium channels in the development of cocaine-associated cardiac arrhythmias
- Lidocaine promotes the trafficking and functional expression of Na v1.8 sodium channels in mammalian cells
- Inhibition of the A-type K + channels of dorsal root ganglion neurons by the long-duration anesthetic butamben
- State-dependent trapping of flecainide in the cardiac sodium channel
- Closing and Inactivation Potentiate the Cocaethylene Inhibition of Cardiac Sodium Channels by Distinct Mechanisms
- Kv1.1 channels of dorsal root ganglion neurons are inhibited by n-butyl-p-aminobenzoate, a promising anesthetic for the treatment of chronic pain
- Cocaine binds to a common site on open and inactivated human heart (Nav 1.5) sodium channels
- Inhibition of HERG potassium channels by cocaethylene: A metabolite of cocaine and ethanol
- Gating properties of Nav1.7 and Nav1.8 peripheral nerve sodium channels
- Inhibition of human ether-a-go-go potassium channels by cocaine
- Characterization of the isoform-specific differences in the gating of neuronal and muscle sodium channels
- A molecular link between activation and inactivation of sodium channels
- Internal block of human heart sodium channels by symmetrical tetra- alkylammoniums
- Evidence for a direct interaction between internal tetra-alkylammonium cations and the inactivation gate of cardiac sodium channels
