900 Walnut Street
Suite 200
Philadelphia, PA 19107
(215) 955-1234
(215) 955-1390 fax
Suite 200
Philadelphia, PA 19107
(215) 955-1234
(215) 955-1390 fax
Most Recent Peer-reviewed Publications
- Low dose zymosan ameliorates both chronic and relapsing experimental autoimmune encephalomyelitis
- Role of serine proteases in inflammation: Bowman-Birk protease inhibitor (BBI) as a potential therapy for autoimmune diseases
- Immune tolerance induced by intravenous transfer of immature dendritic cells via up-regulating numbers of suppressive IL-10+ IFN-γ+-producing CD4+ T cells
- Accelerated and enhanced effect of CCR5-transduced bone marrow neural stem cells on autoimmune encephalomyelitis
- TCR stimulation upregulates MS4a4B expression through induction of AP-1 transcription factor during T cell activation
- CNS-specific therapy for ongoing EAE by silencing IL-17 pathway in astrocytes
- Intravenous tolerance effectively overcomes enhanced pro-inflammatory responses and experimental autoimmune encephalomyelitis severity in the absence of IL-12 receptor signaling
- IL-10 deficiency blocks the ability of LPS to regulate expression of tolerance-related molecules on dendritic cells
- Interleukin-10 plays a crucial role in suppression of experimental autoimmune encephalomyelitis by Bowman-Birk inhibitor
- Bowman-Birk inhibitor attenuates experimental autoimmune encephalomyelitis by delaying infiltration of inflammatory cells into the CNS
- Generation of immunogenic and tolerogenic clinical-grade dendritic cells
- IL-9 is important for T-cell activation and differentiation in autoimmune inflammation of the central nervous system
- Kit (W-sh) mice develop earlier and more severe experimental autoimmune encephalomyelitis due to absence of immune suppression
- The encephalitogenicity of T H 17 cells is dependent on IL-1- and IL-23-induced production of the cytokine GM-CSF
- 3G11 expression in CD4 + T cell-mediated autoimmunity and immune tolerance
- Oral resveratrol reduces neuronal damage in a model of multiple sclerosis
- MS4a4B, a CD20 Homologue in T Cells, Inhibits T Cell Propagation by Modulation of Cell Cycle
- IDO upregulates regulatory T cells via tryptophan catabolite and suppresses encephalitogenic T cell responses in Experimental autoimmune encephalomyelitis
- Neutralization of IL-9 ameliorates experimental autoimmune encephalomyelitis by decreasing the effector T cell population
- Accelerated thymocyte maturation in IL-12Rβ2-deficient mice contributes to increased susceptibility to autoimmune inflammatory demyelination