1020 Sansom Street
Thompson Building, Suite 239
Philadelphia, PA 19107
(215) 955-5105
(215) 503-5686 fax
Thompson Building, Suite 239
Philadelphia, PA 19107
(215) 955-5105
(215) 503-5686 fax
Most Recent Peer-reviewed Publications
- C1q/TNF-related proteins, a family of novel adipokines, induce vascular relaxation through the adiponectin receptor-1/AMPK/eNOS/nitric oxide signaling pathway
- Expectations of an emergency medicine clerkship director
- Triage during the week of the Sichuan earthquake: A review of utilized patient triage, care, and disposition procedures
- Emergency medicine in the medical school curriculum.
- Reduced vascular responsiveness to adiponectin in hyperlipidemic rats-mechanisms and significance
- Thioredoxin glycation: A novel posttranslational modification that inhibits its antioxidant and organ protective actions
- Methylglyoxal increases cardiomyocyte ischemia-reperfusion injury via glycative inhibition of thioredoxin activity
- The dragon strikes: Lessons from the wenchuan earthquake
- Emergency medicine and political influence
- Cardioprotective effect of adiponectin is partially mediated by its AMPK-independent antinitrative action
- Endothelial dysfunction in adiponectin deficiency and its mechanisms involved
- AMP-activated protein kinase deficiency enhances myocardial ischemia/reperfusion injury but has minimal effect on the antioxidant/ antinitrative protection of adiponectin
- Rosiglitazone inhibits hypercholesterolaemia-induced myeloperoxidase upregulation - A novel mechanism for the cardioprotective effects of PPAR agonists
- Reduced cardiotropic response to insulin in spontaneously hypertensive rats: Role of peroxisome proliferator-activated receptor-γ-initiated signaling
- Adiponectin improves endothelial function in hyperlipidemic rats by reducing oxidative/nitrative stress and differential regulation of eNOS/iNOS activity
- TNFα-initiated oxidative/nitrative stress mediates cardiomyocyte apoptosis in traumatic animals
- Tumor necrosis factor-α in mechanic trauma plasma mediates cardiomyocyte apoptosis
- Nitrative thioredoxin inactivation as a cause of enhanced myocardial ischemia/reperfusion injury in the aging heart
- Role of iNOS-derived reactive nitrogen species and resultant nitrative stress in leukocytes-induced cardiomyocyte apoptosis after myocardial ischemia/reperfusion
- Effects of N-n-butyl haloperidol iodide on L-type calcium channels and intracellular free calcium in rat ventricular myocytes