1020 Locust Street
Suite 452
Philadelphia, PA 19107
(215) 503-1774
(215) 923-7745 fax
Suite 452
Philadelphia, PA 19107
(215) 503-1774
(215) 923-7745 fax
Most Recent Peer-reviewed Publications
- Aleuria Aurantia Lectin (AAL)-Reactive Immunoglobulin G Rapidly Appears in Sera of Animals following Antigen Exposure
- Temporal changes in innate immune signals in a rat model of alcohol withdrawal in emotional and cardiorespiratory homeostatic nuclei
- Oxidative stress and reduced glutamine synthetase activity in the absence of inflammation in the cortex of mice with experimental allergic encephalomyelitis
- Rabies Virus Clearance from the Central Nervous System
- The Role of Toll-Like Receptors in the Induction of Immune Responses During Rabies Virus Infection
- Therapeutic immune clearance of rabies virus from the CNS
- Plant vaccines: An immunological perspective
- Modulation of serum uric acid levels by inosine in patients with multiple sclerosis does not affect blood pressure
- IL-4 -/- mice with lethal Mesocestoides corti infections - Reduced Th2 cytokines and alternatively activated macrophages
- Effects of treatments on inflammatory and apoptotic markers in the CNS of mice with globoid cell leukodystrophy
- The production of antibody by invading B cells is required for the clearance of rabies virus from the central nervous system
- Effective preexposure and postexposure prophylaxis of rabies with a highly attenuated recombinant rabies virus
- The treatment of multiple sclerosis with inosine.
- Immune evasion by rabies viruses through the maintenance of blood-brain barrier integrity
- Blood-brain barrier changes and cell invasion differ between therapeutic immune clearance of neurotrophic virus and CNS autoimmunity
- Serum and cerebrospinal fluid uric acid levels in multiple sclerosis patients
- Early blood-brain barrier permeability in cerebella of PLSJL mice immunized with myelin basic protein
- Lethal silver-haired bat rabies virus infection can be prevented by opening the blood-brain barrier
- A peroxynitrite-dependent pathway is responsible for blood-brain barrier permeability changes during a central nervous system inflammatory response: TNF-α is neither necessary nor sufficient
- Loss of blood-brain barrier integrity in the spinal cord is common to experimental allergic encephalomyelitis in knockout mouse models