233 South 10th Street
228 BLSB
Philadelphia, PA 19107
(215) 503-4907
(215) 923-9162 fax
228 BLSB
Philadelphia, PA 19107
(215) 503-4907
(215) 923-9162 fax
Most Recent Peer-reviewed Publications
- Activation of Hsp70 reduces neurotoxicity by promoting polyglutamine protein degradation
- A polyglutamine expansion disease protein sequesters PTIP to attenuate DNA repair and increase genomic instability
- Attacking the flank: Targeting new pathways in SBMA
- Guidelines for the use and interpretation of assays for monitoring autophagy
- Testosterone treatment fails to accelerate disease in a transgenic mouse model of spinal and bulbar muscular atrophy
- SIRT1 modulates aggregation and toxicity through deacetylation of the androgen receptor in cell models of SBMA
- An interdomain interaction of the androgen receptor is required for its aggregation and toxicity in spinal and bulbar muscular atrophy
- Autophagy and access: Understanding the role of androgen receptor subcellular localization in SBMA
- FOXO3a is broadly neuroprotective in vitro and in vivo against insults implicated in motor neuron diseases
- Cytoplasmic retention of polyglutamine-expanded androgen receptor ameliorates disease via autophagy in a mouse model of spinal and bulbar muscular atrophy
- Cyclin D1 repressor domain mediates proliferation and survival in prostate cancer
- ASC-J9 ameliorates spinal and bulbar muscular atrophy phenotype via degradation of androgen receptor
- Soluble androgen receptor oligomers underlie pathology in a mouse model of spinobulbar muscular atrophy
- Animal models of Kennedy disease
- Castration restores function and neurofilament alterations of aged symptomatic males in a transgenic mouse model of spinal and bulbar muscular atrophy
- Transglutaminase potentiates ligand-dependent proteasome dysfunction induced by polyglutamine-expanded androgen receptor
- Ligand promotes intranuclear inclusions in a novel cell model of spinal and bulbar muscular atrophy
- Trinucleotide repeat disease. The androgen receptor in spinal and bulbar muscular atrophy
- Aggregated polyglutamine peptides delivered to nuclei are toxic to mammalian cells
- Polyglutamine and transcription: Gene expression changes shared by DRPLA and Huntington's disease mouse models reveals context-independent effects
